Abstract
In this issue of Molecular Cell, Park et al. (2016) elegantly demonstrate that a partial defect in autophagy supports malignant transformation as it favors the production of genotoxic reactive oxygen species by mitochondria. In this issue of Molecular Cell, Park et al. (2016) elegantly demonstrate that a partial defect in autophagy supports malignant transformation as it favors the production of genotoxic reactive oxygen species by mitochondria.
Original language | English |
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Pages (from-to) | 473-474 |
Number of pages | 2 |
Journal | Molecular Cell |
Volume | 62 |
Issue number | 4 |
DOIs | |
Publication status | Published - 19 May 2016 |
Keywords
- Alternative splicing
- Autophagic flux
- Mutational rate
- Myelodysplastic syndrome
- Regulated cell death
- U2AF1