Peliosis hepatis, nodular regenerative hyperplasia of the liver, and light-chain deposition in a patient with Waldenström's macroglobulinemia

Odile Voinchet; Claude Degott; Jean Yves Scoazec; Gérard Feldmann; Jean Pierre Benhamou

doi:10.1016/0016-5085(88)90508-2

Peliosis hepatis, nodular regenerative hyperplasia of the liver, and light-chain deposition in a patient with Waldenström's macroglobulinemia

Odile Voinchet, Claude Degott, Jean Yves Scoazec, Gérard Feldmann, Jean Pierre Benhamou

Résultats de recherche: Contribution à un journal › Article › Revue par des pairs

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Résumé

We report the case of a female patient suffering from Waldenström's macroglobulinemia with three different liver lesions: peliosis hepatis, nodular regenerative hyperplasia, and light-chain deposits within the sinusoidal walls. We hypothesize that both peliosis hepatis and nodular regenerative hyperplasia might be the consequence of a disordered intrahepatic circulation determined by light-chain deposits infiltrating the sinusoidal walls.

langue originale	Anglais
Pages (de - à)	482-486
Nombre de pages	5
journal	Gastroenterology
Volume	95
Numéro de publication	2
Les DOIs	https://doi.org/10.1016/0016-5085(88)90508-2
état	Publié - 1 janv. 1988

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10.1016/0016-5085(88)90508-2

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abstract = "We report the case of a female patient suffering from Waldenstr{\"o}m's macroglobulinemia with three different liver lesions: peliosis hepatis, nodular regenerative hyperplasia, and light-chain deposits within the sinusoidal walls. We hypothesize that both peliosis hepatis and nodular regenerative hyperplasia might be the consequence of a disordered intrahepatic circulation determined by light-chain deposits infiltrating the sinusoidal walls.",

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AU - Feldmann, Gérard

AU - Benhamou, Jean Pierre

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AB - We report the case of a female patient suffering from Waldenström's macroglobulinemia with three different liver lesions: peliosis hepatis, nodular regenerative hyperplasia, and light-chain deposits within the sinusoidal walls. We hypothesize that both peliosis hepatis and nodular regenerative hyperplasia might be the consequence of a disordered intrahepatic circulation determined by light-chain deposits infiltrating the sinusoidal walls.

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