When Death Was Young: An Ancestral Apoptotic Network in Bacteria

Didac Carmona-Gutierrez; Guido Kroemer; Frank Madeo

doi:10.1016/j.molcel.2012.05.032

When Death Was Young: An Ancestral Apoptotic Network in Bacteria

Didac Carmona-Gutierrez, Guido Kroemer, Frank Madeo

Résultats de recherche: Contribution à un journal › Brève enquête › Revue par des pairs

15 Citations (Scopus)

Résumé

In this issue of Molecular Cell, Dwyer et al. (2012) characterize a RecA-dependent and ClpXP-regulated pathway that controls the acquisition of several apoptotic markers upon bactericidal treatment of prokaryotes, placing the hypothetical origin of apoptosis further downstream in evolution.

langue originale	Anglais
Pages (de - à)	552-554
Nombre de pages	3
journal	Molecular Cell
Volume	46
Numéro de publication	5
Les DOIs	https://doi.org/10.1016/j.molcel.2012.05.032
état	Publié - 8 juin 2012

Accès au document

10.1016/j.molcel.2012.05.032

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title = "When Death Was Young: An Ancestral Apoptotic Network in Bacteria",

abstract = "In this issue of Molecular Cell, Dwyer et al. (2012) characterize a RecA-dependent and ClpXP-regulated pathway that controls the acquisition of several apoptotic markers upon bactericidal treatment of prokaryotes, placing the hypothetical origin of apoptosis further downstream in evolution.",

author = "Didac Carmona-Gutierrez and Guido Kroemer and Frank Madeo",

year = "2012",

month = jun,

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doi = "10.1016/j.molcel.2012.05.032",

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journal = "Molecular Cell",

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T2 - An Ancestral Apoptotic Network in Bacteria

AU - Carmona-Gutierrez, Didac

AU - Kroemer, Guido

AU - Madeo, Frank

PY - 2012/6/8

Y1 - 2012/6/8

N2 - In this issue of Molecular Cell, Dwyer et al. (2012) characterize a RecA-dependent and ClpXP-regulated pathway that controls the acquisition of several apoptotic markers upon bactericidal treatment of prokaryotes, placing the hypothetical origin of apoptosis further downstream in evolution.

AB - In this issue of Molecular Cell, Dwyer et al. (2012) characterize a RecA-dependent and ClpXP-regulated pathway that controls the acquisition of several apoptotic markers upon bactericidal treatment of prokaryotes, placing the hypothetical origin of apoptosis further downstream in evolution.

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U2 - 10.1016/j.molcel.2012.05.032

DO - 10.1016/j.molcel.2012.05.032

M3 - Short survey

AN - SCOPUS:84861995560

SN - 1097-2765

VL - 46

SP - 552

EP - 554

JO - Molecular Cell

JF - Molecular Cell

IS - 5

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